Virus more efficient at infection after mutation; diseased lungs more receptive to virus

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Virus more efficient at infection after mutation; diseased lungs more receptive to virus
日本 最新ニュース,日本 見出し
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The following is a brief roundup of some of the latest scientific studies on the novel coronavirus and efforts to find treatments and vaccines for COVID-19, the illness caused by the virus.

New data helps explain why people with respiratory conditions appear to be more vulnerable to coronavirus infections. The virus breaks into cells via a receptor protein on the cell surface called ACE2. People with conditions like asthma, chronic obstructive pulmonary disease, pulmonary hypertension, and smokers have more of the ACE2 receptors on their lung cells than healthy people do, researchers found.

Under the experimental conditions, potentially contagious virus was still present - although at extremely low levels - after 21 days on a plastic visor, 14 days on stainless steel and Tyvek coveralls, 7 days on nitrile gloves and 4 days on chemical resistant gloves. But only an hour after virus particles were applied to 100% cotton fabric, the amount of active virus had decreased by 99.9%, and it was completely undetectable in less than 24 hours.

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