In recent days, there were several important new reports that help explain the pathophysiology of long COVID. The new findings are summarized here.
In recent days, there were several important new reports that help explain the pathophysiology of long COVID. By coincidence, we published a, using MRI, brain-specific S100β protein, RNA-seq of white blood cells, endothelial cell response in culture provided new insights about the disrupted blood-brain barrier in individuals with long COVID and symptoms of brain fog.
The numbers of participants in each group was small, but the phenotyping was extraordinarily deep. Beyond the BBB, the notable findings were dysregulation of the clotting system and pro-inflammatory effects of endothelial cells., 381 participants were found to have evidence of persistent infections of SARS-CoV-2 for 30 days or more. That was considerably higher than anticipated.
One percent to 3% had persistent infections for more than 30 days. In addition, 0.1%-0.5% had persistent infections for more than 60 days. Serial assessment of viral loads indicated many of the people had rebounding high levels, indicating that there was likely actively replicating virus. The risk for long COVID was increased by 55% in people with 12 weeks or more of persistent infection and by 24% in those with infection for 26 weeks or more., part of the first line of defense of innate immunity to a SARS-CoV-2 infection. Individuals with long COVID had persistent elevation after the acute phase, and during follow-up serial assessment of this biomarker showed resolution among the participants with symptom resolution.
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