There’s reason to be hopeful: 'The coronavirus is starting to look a lot like other viruses that have led to good vaccines,' said one expert.
Typically, T cells and B cells hang around after an infection, primed to rapidly respond to a reinvasion by making more antibodies. And there are indications that after recovery from COVID-19, they should be long-lasting: In the closest human-infecting relative to the novel coronavirus, SARS, awho recovered from that virus in 2003 showed they still had memory T cells for the virus 17 years later.
“The devil is in the details,” Altmann said. “We need to keep watching over several months and years.”Not at all. Mild cases seem to produce fewer antibodies while more severe ones make more of them. In one Chinese study, for example, 40% of asymptomatic patients hadeight weeks after an infection. That same paradox, a seemingly weaker antibody response in milder cases, happened with SARS.
“We had patients of all three types die, even the third immunotype, but the first group was clearly the worst situation,” said Wherry. This kind of “heterogeneity” in immune system responses to a virus is unusual, he added. “We are all scratching our heads over this,” said Wherry.
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